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Correction: Hyperoside, a dietary flavonoid, protects against endometritis via gut microbiota-dependent production of hydroxyphenyllactic acid and the gut–uterus axis
Jing Yang†
a,
Jing Yu†a,
Yajing Chen†a,
Anqi Xuab,
Chunli Yanga,
Jincun Liac,
Fengkun Wuac,
Xiaobing Lia,
Junlong Bi*a,
Bin Xiang*a and
Kangfeng Jiang*a
aCollege of Veterinary Medicine, Yunnan Agricultural University, Kunming, 650201 Yunnan, China. E-mail: kangfengjiang@ynau.edu.cn; xiangbin2018@126.com; junlongbi@foxmail.com
bZhaotong Municipal Animal Health Supervision Institute, Zhaotong, 657000 Yunnan, China
cXundian County Agriculture and Rural Bureau, Xundian, 655200 Yunnan, China
First published on 16th February 2026
Abstract
Correction for ‘Hyperoside, a dietary flavonoid, protects against endometritis via gut microbiota-dependent production of hydroxyphenyllactic acid and the gut–uterus axis’ by Jing Yang, et al., Food Funct., 2026, 17, 408–425, https://doi.org/10.1039/D5FO04275E.
The authors regret that in the above article Fig. 1 was incorrectly presented. The correct Fig. 1 is shown here.
 | ||
| Fig. 1 Hyperoside attenuated E. coli-induced endometritis and intestinal inflammation in mice. (A) Illustration of the in vivo mouse experiment. The mice were divided into four groups and injected with 108 CFU of E. coli in the uterus to induce endometritis and then treated with hyperoside. Created with BioGDP.com.64 (B) Uterine tissue morphology. (C and D) H&E staining of the mice uterine tissue samples and histological scores of uterine tissues (n = 3 per group). (E–G) The expression levels of tight junction proteins occludin and ZO-1 were measured by immunofluorescence. Occludin protein was labeled with a green fluorophore, ZO-1 protein was labeled with a red fluorophore, and the cell nucleus was labeled with a blue fluorophore (n = 5 per group). (H–J) The expression levels of TNF-α, IL-1β and IL-6 were measured by RT-qPCR (n = 3 per group). (K–M) The expression levels of TLR4 and p-p65 were detected by western blotting (n = 3 per group). β-Actin was used as a control. (N) Colon morphology. (O) Colon length (n = 4 per group). (P and Q) H&E staining of the mice colon tissue samples and histological scores of colonic tissues (n = 3 per group). Data are presented as the mean ± SEM of three independent experiments. Statistical significance was determined using one-way ANOVA. *p < 0.05, **p < 0.01, and ***p < 0.001. | ||
The results and conclusions of the study remain the same and are unaffected by this change.
The Royal Society of Chemistry apologises for these errors and any consequent inconvenience to authors and readers.
Footnote |
| † These authors have contributed equally to this work. |
| This journal is © The Royal Society of Chemistry 2026 |