Borrelidin modulates the alternative splicing of VEGF in favour of anti-angiogenic isoforms†
Abstract
The
* Corresponding authors
a
Biotica, Chesterford Research Park, Cambridge, UK
E-mail:
barrie.wilkinson@biotica.com
Fax: +44 (0)1799 532921
Tel: +44 (0)1799 532925
b
Microvascular Research Laboratories, Bristol Heart Institute, Department of Physiology and Pharmacology, School of Veterinary Sciences, University of Bristol, Southwell Street Bristol, UK
E-mail:
dave.bates@bristol.ac.uk
Fax: +44 (0)117 9288151
Tel: +44 (0)117 9289818
c Department of Biochemistry, University of Cambridge, 80 Tennis Court Road, Cambridge, UK
d School of Chemistry and Manchester Interdisciplinary Biocentre, University of Manchester, 131 Princess Street, Manchester, UK
e The University of Texas M.D. Anderson Cancer Centre, Science Park-Research Division, Smithville, Texas, USA
The
J. Woolard, W. Vousden, S. J. Moss, A. Krishnakumar, M. V. R. Gammons, D. G. Nowak, N. Dixon, J. Micklefield, A. Spannhoff, M. T. Bedford, M. A. Gregory, C. J. Martin, P. F. Leadlay, M. Q. Zhang, S. J. Harper, D. O. Bates and B. Wilkinson, Chem. Sci., 2011, 2, 273 DOI: 10.1039/C0SC00297F
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