Reply to Comment on “Deletion of btn1, an orthologue of CLN3, increases glycolysis and perturbs amino acid metabolism in the fission yeast model of Batten disease”
Abstract
We respond briefly to the letter received from Pearce and Padilla-Lopez.
* Corresponding authors
a
MRC Laboratory for Molecular Cell Biology, University College London, Gower Street, London, UK
E-mail:
s.mole@ucl.ac.uk
b Molecular Medicine Unit, UCL Institute of Child Health, University College London, UK
c Department of Genetics, Evolution and Environment, University College London, Gower Street, London, UK
d Department of Biochemistry, University of Cambridge, Hopkins Building, Tennis Court Road, Cambridge, UK
e The Cambridge Centre for Systems Biology, University of Cambridge, Hopkins Building, Tennis Court Road, Cambridge, UK
We respond briefly to the letter received from Pearce and Padilla-Lopez.
S. E. Mole, S. Codlin and J. L. Griffin, Mol. BioSyst., 2011, 7, 1349 DOI: 10.1039/C0MB00300J
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