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Alzheimer's disease: which type of amyloid-preventing drug agents to employ?

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Corresponding authors
a
Center for Cancer Research, Nanobiology Program, Basic Science Program, SAIC-Frederick, Inc., National Cancer Institute, Frederick, USA
E-mail: nussinor@helix.nih.gov
b
Departments of Bioengineering and of Mechanical and Aerospace Engineering, and Materials Science Program, University of California, San Diego, La Jolla, USA
c
Department of Psychiatry, David Geffen School of Medicine, Semel Institute for Neuroscience and Human Behavior, University of California, Los Angeles, USA
E-mail: bkagan@mednet.ucla.edu
d
Department of Human Molecular Genetics and Biochemistry, Sackler School of Medicine, Tel Aviv University, Tel Aviv 69978, Israel
Phys. Chem. Chem. Phys., 2013,15, 8868-8877

DOI: 10.1039/C3CP00017F
Received 02 Jan 2013, Accepted 20 Feb 2013
First published online 21 Feb 2013

This article is part of themed collection: Biophysical studies on protein misfolding and amyloid diseases
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