Issue 4, 2011
From the journal:

Molecular BioSystems

Reply to Comment on “Deletion of btn1, an orthologue of CLN3, increases glycolysis and perturbs amino acid metabolism in the fission yeast model of Batten disease”

Sara E. Mole,abc   Sandra Codlina  and  Julian L. Griffin*de  

* Corresponding authors

a MRC Laboratory for Molecular Cell Biology, University College London, Gower Street, London, UK
E-mail: s.mole@ucl.ac.uk

b Molecular Medicine Unit, UCL Institute of Child Health, University College London, UK

c Department of Genetics, Evolution and Environment, University College London, Gower Street, London, UK

d Department of Biochemistry, University of Cambridge, Hopkins Building, Tennis Court Road, Cambridge, UK

e The Cambridge Centre for Systems Biology, University of Cambridge, Hopkins Building, Tennis Court Road, Cambridge, UK

Abstract

We respond briefly to the letter received from Pearce and Padilla-Lopez.

Graphical abstract: Reply to Comment on “Deletion of btn1, an orthologue of CLN3, increases glycolysis and perturbs amino acid metabolism in the fission yeast model of Batten disease”

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Article information

DOI
https://doi.org/10.1039/C0MB00300J
Article type
Letter
Submitted
25 Nov 2010
Accepted
15 Dec 2010
First published
24 Jan 2011

Mol. BioSyst., 2011,7, 1349-1349

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Reply to Comment on “Deletion of btn1, an orthologue of CLN3, increases glycolysis and perturbs amino acid metabolism in the fission yeast model of Batten disease”

S. E. Mole, S. Codlin and J. L. Griffin, Mol. BioSyst., 2011, 7, 1349 DOI: 10.1039/C0MB00300J

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